Cyril MARTIN
Dr. Cyril Martin is an exercise physiologist, Associate Professor at Claude Bernard University in Lyon. After graduating in 2000 from the University of Bordeaux, Cyril held a lecturer position at University of Bordeaux (2000-2002). He moved in 2002 as an Assistant Professor at University of Lyon. He was graduate studies director (2003-2011) at UFR STAPS in Lyon, and since 2011 is director of Master STAPS « Entraînement et Optimisation de la Performance Sportive » of University of Lyon 1. After defending his PhD dealing with neuroendocrinology of physical exercise, Cyril developed between 2002 and 2019 researches on the effects of acute and/or habitual exercise in sickle cell trait and sickle cell anemia, notably focusing on inflammatory, hemorheological, oxidative stress parameters. He demonstrated that habitual physical activity favorably modulates inflammation, oxidative stress and blood rheology in subjects with sickle cell trait, as well as in sickle cell mice models and patients with sickle cell anemia. His current research work focuses on the optimization of training and conditioning methods in different sports, with a particular focus targeting application in female athletes (impact of the menstrual cycle on performance factors). All of these research activities have received broad financial support (over € 1,200,000, including over € 800,000 as principal investigator). To carry out this work, Cyril supervised 7 PhD students and 2 post-doctoral fellows. As of November 2020, Cyril had published 67 articles in ISI indexed journals.
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Cyril Martin (cyril.martin @ univ-lyon1.fr)
Institution Université Claude Bernard (Lyon 1) Team: [SPIP] Position Associate Professor
Publications |
1. The sensitivity of the corticotropic axis determines the physiological responses to stress and physical training. 2. Carriers of the skickle-cell trait present haemorheologic, inflammatory and oxydative alterations in response to intense exercise. 3. Regular physical activity favourably modulates haemorheologic, inflammatory and oxydative responses in carriers of the skickle-cell trait. 4. Training in mice with the sickle-cell homozygote limits endothelial activation, oxydative stress, and rheologic alterations. |